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Wnt/β-catenin signaling pathway in trophoblasts and abnormal activation in preeclampsia (Review)

Mol Med Rep. 2017 Aug;16(2):1007-1013. doi: 10.3892/mmr.2017.6718. Epub 2017 Jun 7.

Abstract

Preeclampsia (PE) is one of the most common types of hypertensive disease and occurs in 3‑4% of pregnancies. There are a number of theories on the pathogenesis of PE. Abnormal differentiation of the placenta may lead to failure of trophoblast migration, shallow placenta implantation and placental ischemia/hypoxia, followed by the subsequent occurrence of PE. The Wnt/β-catenin pathway is a canonical Wnt‑signaling pathway that regulates several biological processes, including proliferation, migration, invasion and apoptosis. Abnormal activation of the Wnt/β‑catenin signaling pathway may serve an important role in the pathogenesis of various human diseases, particularly in human cancer. Recent studies have demonstrated that the dysregulation of the Wnt/β‑catenin signaling pathway may contribute to PE. The present review aims to summarize the articles on Wnt/β‑catenin signaling pathway in the trophoblast and abnormal activation in PE. Wnt/β-catenin signaling may serve a significant role in the pathogenesis of PE and may be a prospective therapeutic target for the prevention and treatment of PE.

Publication types

  • Review

MeSH terms

  • Female
  • Frizzled Receptors / metabolism
  • Humans
  • Placenta / metabolism
  • Pre-Eclampsia / metabolism
  • Pre-Eclampsia / pathology*
  • Pregnancy
  • Trophoblasts / metabolism*
  • Wnt Proteins / chemistry
  • Wnt Proteins / metabolism
  • Wnt Signaling Pathway / physiology*
  • beta Catenin / metabolism

Substances

  • Frizzled Receptors
  • Wnt Proteins
  • beta Catenin