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Activation of the alternative pathway of human complement by haemoglobin

Clin Exp Immunol. 1979 Apr;36(1):140-4.

Abstract

Haemoglobin solutions (concentration greater than 1.5 mg/ml), prepared from lysates of erythrocytes from a normal subject and from a patient with sickle cell anaemia, caused factor B and C3 cleavage and loss of haemolytic activity of factor B wehn incubated with fresh autologous serum. Under the same experimental conditions, preparations of erythrocyte stroma or of buffycoat lysates did not produce factor B and C3 cleavage. This reaction required Mg++ but not C1q or C4, indicating that the alternative complement pathway was activated.

MeSH terms

  • Anemia, Sickle Cell / immunology
  • Calcium / blood
  • Complement Activation* / drug effects
  • Complement C3 / metabolism
  • Complement Factor B / metabolism
  • Complement Pathway, Alternative* / drug effects
  • Egtazic Acid / pharmacology
  • Hemoglobins / immunology*
  • Hemolysis
  • Humans
  • Immunoelectrophoresis
  • Magnesium / blood

Substances

  • Complement C3
  • Hemoglobins
  • Egtazic Acid
  • Complement Factor B
  • Magnesium
  • Calcium