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Reconstitution of H-2 class I expression by gene transfection decreases susceptibility to natural killer cells of an EL4 class I loss variant. (Q54337080)

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scientific article published in January 1990

Language	Label	Description	Also known as
English	Reconstitution of H-2 class I expression by gene transfection decreases susceptibility to natural killer cells of an EL4 class I loss variant.	scientific article published in January 1990

Statements

scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

28 May 2018

http://europepmc.org/abstract/MED/2106441

Reconstitution of H-2 class I expression by gene transfection decreases susceptibility to natural killer cells of an EL4 class I loss variant. (English)

1 reference

Europe PubMed Central

PubMed publication ID

28 May 2018

http://europepmc.org/abstract/MED/2106441

author name string

Sturmhöfel K

1

1 reference

Europe PubMed Central

PubMed publication ID

28 May 2018

http://europepmc.org/abstract/MED/2106441

Hämmerling GJ

2

1 reference

Europe PubMed Central

PubMed publication ID

28 May 2018

http://europepmc.org/abstract/MED/2106441

language of work or name

0 references

publication date

1 January 1990

1 reference

Europe PubMed Central

PubMed publication ID

28 May 2018

http://europepmc.org/abstract/MED/2106441

European Journal of Immunology

1 reference

Europe PubMed Central

PubMed publication ID

28 May 2018

http://europepmc.org/abstract/MED/2106441

20

1 reference

Europe PubMed Central

PubMed publication ID

28 May 2018

http://europepmc.org/abstract/MED/2106441

1

1 reference

Europe PubMed Central

PubMed publication ID

28 May 2018

http://europepmc.org/abstract/MED/2106441

171-177

1 reference

Europe PubMed Central

PubMed publication ID

28 May 2018

http://europepmc.org/abstract/MED/2106441

Mechanism of cytotoxicity by natural killer (NK) cells

1 reference

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Lung colonization and metastasis by disseminated B16 melanoma cells: H-2 associated control at the level of the host and the tumor cell

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Selective rejection of H-2-deficient lymphoma variants suggests alternative immune defence strategy.

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Host H-2 genotype regulates the metastatic ability of H-2-associated variants of B16 melanoma: defense systems screening for absence of self H-2 components by natural killer cells and host-associated homing barrier.

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Natural killer susceptibility of human cells may be regulated by genes in the HLA region on chromosome 6.

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Interferon-induced alterations in metastatic capacity, class-1 antigen expression and natural killer cell sensitivity of melanoma cells

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Demonstration by class I gene transfer that reduced susceptibility of human cells to natural killer cell-mediated lysis is inversely correlated with HLA class I antigen expression

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Reversal of natural killing susceptibility in target cells expressing transfected class I HLA genes

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Fine Specificity Analysis with Monoclonal Antibodies of Antigens Controlled by the Major Histocompatibility Complex and by the Qa/TL Region in Mice

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Two allelic forms of mouse beta 2-microglobulin

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Electrotransfection of human lymphoid and myeloid cell lines

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New cosmid vectors developed for eukaryotic DNA cloning

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Domain interactions of H–2 class I antigens alter cytotoxic T-cell recognition sites

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Insulin binding to human B lymphoblasts is a function of HLA haplotype

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Influence of the mouse major histocompatibility complex, H-2, on liver adenylate cyclase activity and on glucagon binding to liver cell membranes

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Structure of the human class I histocompatibility antigen, HLA-A2

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Substitution at residue 227 of H-2 class I molecules abrogates recognition by CD8-dependent, but not CD8-independent, cytotoxic T lymphocytes

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Identifiers

10.1002/EJI.1830200125

1 reference

Europe PubMed Central

PubMed publication ID

28 May 2018

http://europepmc.org/abstract/MED/2106441

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

28 May 2018

http://europepmc.org/abstract/MED/2106441

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