Cell metabolism in the regulation of programmed cell death (Q34537642)

2 August 2017

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title

Cell metabolism in the regulation of programmed cell death (English)

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2 August 2017

David R Plas

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2 August 2017

Craig B Thompson

series ordinal

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2 August 2017

publication date

1 March 2002

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Trends in Endocrinology and Metabolism

2 August 2017

published in

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2 August 2017

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2 August 2017

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2 August 2017

page(s)

75-78

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Social controls on cell survival and cell death

2 August 2017

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The release of cytochrome c from mitochondria during apoptosis of NGF-deprived sympathetic neurons is a reversible event

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Growth factors can influence cell growth and survival through effects on glucose metabolism

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Outer mitochondrial membrane permeability can regulate coupled respiration and cell survival

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Bcl-xL prevents cell death following growth factor withdrawal by facilitating mitochondrial ATP/ADP exchange

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Mitochondria as the central control point of apoptosis

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In the absence of extrinsic signals, nutrient utilization by lymphocytes is insufficient to maintain either cell size or viability

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Bcl-xL Regulates the Membrane Potential and Volume Homeostasis of Mitochondria

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Bok is a pro-apoptotic Bcl-2 protein with restricted expression in reproductive tissues and heterodimerizes with selective anti-apoptotic Bcl-2 family members

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The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues

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7 January 2021

Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death

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BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and Bak

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The regulation and activities of the multifunctional serine/threonine kinase Akt/PKB.

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Oncogenic kinase signalling

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PTEN, a putative protein tyrosine phosphatase gene mutated in human brain, breast, and prostate cancer

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Phosphatidylinositol 3-kinase signaling mediates angiogenesis and expression of vascular endothelial growth factor in endothelial cells

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A retroviral oncogene, akt, encoding a serine-threonine kinase containing an SH2-like region

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Oncogenic transformation induced by membrane-targeted Akt2 and Akt3.

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Caenorhabditis elegans Akt/PKB transduces insulin receptor-like signals from AGE-1 PI3 kinase to the DAF-16 transcription factor

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7 January 2021

Protein kinase B (PKB/Akt)--a key regulator of glucose transport?

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7 January 2021

Akt and Bcl-xL promote growth factor-independent survival through distinct effects on mitochondrial physiology

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7 January 2021

Inhibition of early apoptotic events by Akt/PKB is dependent on the first committed step of glycolysis and mitochondrial hexokinase

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7 January 2021

Cellular survival: a play in three Akts

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7 January 2021

Akt Phosphorylation of BAD Couples Survival Signals to the Cell-Intrinsic Death Machinery

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7 January 2021

Akt promotes cell survival by phosphorylating and inhibiting a Forkhead transcription factor

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7 January 2021

Expression of the pro-apoptotic Bcl-2 family member Bim is regulated by the forkhead transcription factor FKHR-L1

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7 January 2021

Induction of NF-kappaB by the Akt/PKB kinase

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7 January 2021

NF-kappaB activation by tumour necrosis factor requires the Akt serine-threonine kinase

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7 January 2021

NF-kappaB is a target of AKT in anti-apoptotic PDGF signalling

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7 January 2021

Negative regulation of PKB/Akt-dependent cell survival by the tumor suppressor PTEN

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7 January 2021

Impaired Fas response and autoimmunity in Pten+/- mice

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7 January 2021

Effect of haematopoietic cell growth factor on intracellular ATP levels.

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7 January 2021

10.1016/S1043-2760(01)00528-8

Identifiers

DOI

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2 August 2017

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