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Effects of chitosan and oligochitosans on the phosphatidylinositol 3-kinase-AKT pathway in cancer therapy

Int J Biol Macromol. 2020 Dec 1:164:456-467. doi: 10.1016/j.ijbiomac.2020.07.137. Epub 2020 Jul 18.

Abstract

Phosphatidylinositol 3-kinase (PI3K)-AKT pathway is one of the most important kinase signaling networks in the context of cancer development and treatment. Aberrant activation of AKT, the central mediator of this pathway, has been implicated in numerous malignancies including endometrial, hepatocellular, breast, colorectal, prostate, and, cervical cancer. Thus regulation and blockage of this kinase and its key target nodes is an attractive approach in cancer therapy and diverse efforts have been done to achieve this aim. Chitosan is a carbohydrate with multiple interesting applications in cancer diagnosis and treatment strategies. This bioactive polymer and its derivative oligomers commonly used in drug/DNA delivery methods due to their functional properties which improve efficiency of delivery systems. Further, these compounds exert anti-tumor roles through the stimulation of apoptosis, immune enhancing potency, anti-oxidative features and anti-angiogenic roles. Due to the importance of PI3K-AKT signaling in cancer targeting and treatment resistance, this review discusses the involvement of chitosan, oligochitosaccharides and carriers based on these chemicals in the regulation of this pathway in different tumors.

Keywords: Cancer; Chitosan; Delivery system; Oligochitosans; PI3K-AKT.

Publication types

  • Review

MeSH terms

  • Cell Survival / drug effects
  • Chitin / analogs & derivatives*
  • Chitin / chemistry
  • Chitin / pharmacology
  • Chitin / therapeutic use
  • Chitosan / chemistry
  • Chitosan / pharmacology*
  • Chitosan / therapeutic use
  • Drug Carriers / chemistry
  • Gene Expression Regulation, Neoplastic / drug effects
  • Humans
  • Neoplasms / drug therapy*
  • Neoplasms / metabolism
  • Oligosaccharides
  • Phosphatidylinositol 3-Kinase / metabolism
  • Proto-Oncogene Proteins c-akt / metabolism
  • Signal Transduction / drug effects*

Substances

  • Drug Carriers
  • Oligosaccharides
  • oligochitosan
  • Chitin
  • Chitosan
  • Phosphatidylinositol 3-Kinase
  • Proto-Oncogene Proteins c-akt